特聘教授
肥胖和伴隨的糖尿病成為繁榮時代的重要災難之一。EON体育4平台嘗試從人類進化的角度，研究人類基因組變化🚣♀️⛹🏿，生活方式的改變等對肥胖和糖尿病形成的調控作用。
間充質幹細胞的衰老是肌體許多衰老疾病的細胞學基礎👼🏿🚢。清理衰老細胞(senolysis)可以作為治療衰老相關疾病的重要策略。EON体育4平台研究發現Foxp1基因調控骨髓間充質幹細胞的衰老，這有可能作為調控衰老疾病的重要靶點之一。
以基因敲除小鼠為模型，研究成骨細胞和破骨細胞分化，骨質重塑的調控過程👳🏼，分析骨質疏松，骨關節炎等退行性疾病的病理機製，為治療這些疾病探尋藥物靶點。
Foxp2 regulates anatomical features that may be relevant for vocal behavior and bipedal locomotion.
Foxp1 controls cell fate commitment and senescence of mesenchymal stem cells during skeletal aging
Bone marrow fibrosis with fibrocytic and immunoregulatory responses induced by β-catenin activation in osteoprogenitors
Ablation of Wntless in endosteal niches impairs lymphopoiesis rather than HSC maintenance.
Ectodermal Wnt signaling regulates abdominal myogenesis during ventral body wall development.
Osteoblastic Wnts differentially regulate bone remodeling and the maintenance of bone marrow mesenchymal stem cells.
Wnts-mediated reciprocal regulation between cartilage and bone development during endochondral ossification.
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